By Professor Dr. Gianni Boris Bradač, Priv.-Doz. Dr. Roland Oberson (auth.)
In this age after we are witnessing a veritable explosion in new modalities in diagnos tic imaging we proceed to have a very good want for special reports of the vascularity of the mind in sufferers who've all kinds of cerebral vascular ailment. a lot of the certainty of cerebral vascular occlusive lesions which we constructed within the final 20 years was once in line with our skill to illustrate the vessels that have been affected. a lot experimental paintings in animals have been performed the place significant cerebral vessels have been obstructed and the results of those obstructions at the mind saw pathologically. even if, it was once now not until eventually cerebral angiography can be played with the element that grew to become attainable within the a long time of the '60 's and for this reason that lets start to comprehend the connection of the obstructed vessels saw angiographically to the medical findings. furthermore, a lot physiologic info was once bought. for example, the concept that ofluxury perfusion that is used to explain non-nutritional stream during the tissues was once saw first angiographically even though the time period used to be now not used till LASSEN defined it as a pathophysiological phenomenon saw in the course of cerebral blood stream reviews with radioactive isotopes. the idea that of embolic occlusions of the cerebral vessels as opposed to thrombosis was once clarified and the relative frequency of thrombosis as opposed to embolism was once larger understood. the concept that of collateral move of the mind via so-called meningeal end-to finish arterial anastomoses was once significantly higher understood whilst serial angiography in obstructive cerebral vascular disorder was once performed with expanding frequency.
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Extra resources for Angiography and Computed Tomography in Cerebro-Arterial Occlusive Diseases
Stenosis of this vessel (~) at its origin. Slight arteriosclerotic changes 50 « ) are also visible in the internal carotid artery. -). Arteriosclerotic changes involving the left and right subclavian ( ~) and the left vertebral (_) arteries are also present 51 a 52 b Fig. 17 a-c. Stenosis and occlusion of the vertebral arteries. Fifty-years-old man with TIAs in the vertebrobasilar territory characterized by vertigo, nausea, and ataxia. a Aortic arch angiogram. Early phase. Stenosis of the right vertebral artery (_).
It is surprising how frequently other "secondary lesions" are found together with the clinically suspected lesion on the angiogram. A careful description of all lesions is mandatory since in some cases only the multiplicity of lesions gives a clue to the symptomatology of the patient (Figs. 17, 18, 22, 23). On the other hand, lesions may be present and sometimes may also be very extensive without corresponding clinical symptoms (Figs. 14, 15, 16,23). 5 Tortuosity In accordance with the description of WEIBEL and FIELDS (1965a, b, 1969) the elongation of the internal carotid artery is classified according to the following groups: Tortuosity: S- or C-shaped elongation Coiling: Exaggerated S-shaped or circular curve (Fig.
A Small ulcerative nonstenotic lesion (~). b Late phase: stain of the contrast medium (~). c Another case of small ulcerative nonstenotic lesion (~) III the ulcers 35 a b Fig. 7 a -c. Three examples of occlusion of the internal carotid artery due to atherosclerosis. The different pattern can be explained by the level of the atherothrombotic lesion. The stump of the occluded internal carotid artery, particularly if irregular (c), may be the site of atheroma and/or thrombus which are the potential 36 source of emboli reaching the ophthalmic and/or intracranial circulation by way of the external carotid artery.
Angiography and Computed Tomography in Cerebro-Arterial Occlusive Diseases by Professor Dr. Gianni Boris Bradač, Priv.-Doz. Dr. Roland Oberson (auth.)